TTE Cheatsheet

Severity colours: Normal Mild Moderate Severe. Values follow the current guideline named in each section heading (checked September 2026). Always combine several parameters. Open the calculators with the button at the bottom right.

Standard views & what to measure

ViewWalls / structures seenMeasure
PLAXAnteroseptum (basal–mid) · inferolateral wall · MV, AV, LVOT, aortic root, LALVIDd/s, IVSd, LVPWd (chordal level, end-diastole) · LVOT diameter (mid-systole, ~0.5–1 cm below annulus) · aortic root/sinus/STJ/asc Ao · LA AP diameter · RVOT prox
RV inflowRV, TV (anterior & posterior leaflets), RATR colour/CW
PSAX – AV levelAV cusps, RVOT, PV, PA, TV, LA, IASAV morphology (tri/bicuspid) · RVOT distal · PA diameter · PV PW/CW (AcT) · PR · TR
PSAX – MV levelBasal 6 segments · MV fish-mouthMVA planimetry · scallop localisation
PSAX – papillary levelMid 6 segmentsWall motion · septal flattening / eccentricity index
PSAX – apicalApical 4 segmentsWall motion · apical thrombus
A4CInferoseptum · anterolateral wall · apex · all 4 chambersBiplane Simpson (with A2C) · LA/RA volume/area · RV basal/mid/long dims · TAPSE · RV S' · FAC · MV inflow E/A, DT · e' septal & lateral · TR CW
A5CLVOT, AVLVOT PW VTI · AV CW (Vmax, mean gradient, VTI) · AR
A2CInferior · anterior walls · LAABiplane Simpson · LA volume
A3C / APLAXAnteroseptum · inferolateral wall · LVOT/AVAV CW · MR/AR · LVOT gradient (HCM)
RV-focused A4CRV free wall · lateral TV annulusRV basal (>41 dilated), mid, length · FAC · RV strain
SubcostalIAS · RV free wall · IVC · pericardium · hepatic veinsRV wall thickness · IVC diameter & collapse · effusion · hepatic vein flow
SuprasternalArch, descending aortaCoarctation · desc. aorta PW (holodiastolic reversal in AR)
Right parasternal view is often needed to get the true AV peak velocity in AS — use the Pedoff (non-imaging) probe from apex, right parasternal and suprasternal windows.

Left ventricle ASE/EACVI 2015

Linear dimensions (PLAX, end-diastole)

ParameterMenWomen
LVIDd42–58 mm38–52 mm
LVIDs25–40 mm22–35 mm
LVIDd / BSA22–30 mm/m²23–31 mm/m²
IVSd / PWd6–10 mm6–9 mm
Fractional shortening25–43 %27–45 %
RWT = 2·PWd / LVIDd0.24–0.42 (>0.42 = concentric)
LV mass index (linear)49–115 g/m²43–95 g/m²

LV dilatation (LVIDd)

NormalMildModerateSevere
Men42–5859–6364–68>68 mm
Women38–5253–5657–61>61 mm

Wall thickness (LVH severity, IVS/PW)

NormalMildModerateSevere
Men6–1011–1314–16≥17 mm
Women6–910–1213–15≥16 mm

LV geometry

LV mass index normalLV mass index ↑
RWT ≤ 0.42Normal geometryEccentric hypertrophy
RWT > 0.42Concentric remodellingConcentric hypertrophy

Volumes (biplane Simpson)

ParameterMenWomen
EDV62–150 mL46–106 mL
ESV21–61 mL14–42 mL
EDV index≤ 74 mL/m²≤ 61 mL/m²
ESV index≤ 31 mL/m²≤ 24 mL/m²

Systolic function

LVEFNormalMildly ↓Moderately ↓Severely ↓
Men52–72 %41–51 %30–40 %<30 %
Women54–74 %41–53 %30–40 %<30 %

HF classification (LVEF)

HFrEF ≤ 40 % · HFmrEF 41–49 % · HFpEF ≥ 50 % · HFimpEF: baseline ≤40 % with ≥10-point rise to >40 %

Global longitudinal strain

Normal ≈ −20 %. More negative than −18 % likely normal; less negative than −16 % abnormal (vendor-dependent). >15 % relative drop = cardiotoxicity flag.

Other

SV index normal ≥ 35 mL/m² · LVOT VTI ~18–22 cm (<15 cm = low output) · MR dP/dt >1200 mmHg/s normal, <1000 impaired

17-segment model & coronary territories

LADRCALCx
1 Normal2 Hypo3 Akinetic4 Dyskinetic

Top = anterior · left = septum · right = lateral · bottom = inferior

Tap a segment for its name, territory and the views that show it. In wall-motion scoring mode each tap cycles the score (1→2→3→4).
LevelSegments
Basal (1–6)1 anterior · 2 anteroseptal · 3 inferoseptal · 4 inferior · 5 inferolateral · 6 anterolateral
Mid (7–12)7 anterior · 8 anteroseptal · 9 inferoseptal · 10 inferior · 11 inferolateral · 12 anterolateral
Apical (13–16)13 anterior · 14 septal · 15 inferior · 16 lateral
Apex (17)17 apical cap
ArteryTypical segments
LAD1, 2, 7, 8, 13, 14, 17 (± 16 in wrap-around LAD)
RCA3, 4, 9, 10, 15 (± 5, 11 if right-dominant)
LCx5, 6, 11, 12, 16

Which walls in which view

ViewWall 1Opposite wall
PLAXAnteroseptal (2, 8)Inferolateral (5, 11)
A4CInferoseptal (3, 9, 14)Anterolateral (6, 12, 16) + apex 17
A2CInferior (4, 10, 15)Anterior (1, 7, 13) + apex 17
A3C / APLAXInferolateral (5, 11)Anteroseptal (2, 8) + apex 17
PSAX (base/mid/apex)All 6 (or 4) segments at that level — best view to confirm territory

Wall-motion score

1 normal/hyperkinetic · 2 hypokinetic · 3 akinetic · 4 dyskinetic (aneurysm scored as dyskinetic in current ASE). WMSI = sum ÷ segments scored; normal = 1.0.

Definitions

Hypokinetic: reduced thickening (<40 %) · Akinetic: <10 % thickening · Dyskinetic: systolic outward motion/thinning. Thin (<6 mm), bright wall = scar.

Pattern clues

Regional abnormality not following one territory → think takotsubo (apical ballooning), myocarditis, sarcoid (basal septum), LBBB/pacing (septal flash).

Walls by view standard display: apex at top of screen for apical views

Tap any segment: it lights up in every view where it appears (and on the bull's-eye above). Tap again to clear. Colours = LAD · RCA · LCx.

PLAX

Only basal & mid · apex usually off-screen (left)

Top: anteroseptal 2 · 8Bottom: inferolateral 5 · 11

PSAX — basal (mitral valve)

Fish-mouth MV · segments 1–6

Clockwise from 12: anterior → anterolateral → inferolateral → inferior → inferoseptal → anteroseptal

PSAX — mid (papillary muscles)

Segments 7–12 · best level for global wall motion

AL-PM: dual supply (LAD diag + LCx) · PM-PM: single (RCA or LCx) → rupture-prone

PSAX — apical

Below papillary muscles · 4 segments 13–16

Anterior 13 · lateral 16 · inferior 15 · septal 14

A4C

RV on the left of the screen, LV on the right

Left wall: inferoseptal 3 · 9 · 14Right wall: anterolateral 6 · 12 · 16

A2C

From A4C rotate ~60° counter-clockwise · RV disappears, LAA appears

Left wall: inferior 4 · 10 · 15Right wall: anterior 1 · 7 · 13

A3C / apical long axis

Rotate another ~60° · LVOT and aorta appear on the right

Left wall: inferolateral 5 · 11Right wall: anteroseptal 2 · 8

The grey apical parts in A3C fall between the four apical segments. Assign them using A4C and A2C.

Commonly confused walls

PairHow to tell them apartArtery
Anteroseptal vs inferoseptalAnteroseptal is the septum that runs into the aortic root / LVOT (PLAX top wall, A3C right wall). Inferoseptal is the septum next to the RV and tricuspid valve (A4C left wall).LAD vs RCA (PDA)
Anterolateral vs inferolateralAnterolateral = lateral wall in A4C. Inferolateral (old name "posterior") = PLAX bottom wall and A3C left wall, behind the mitral valve, next to the LA / descending aorta.LCx (+ LAD diagonals) vs LCx (± RCA)
Anterior vs anterolateralAnterior = right wall in A2C (on the LAA side), 12 o'clock in PSAX. Anterolateral = right wall in A4C, 2 o'clock in PSAX.LAD vs LCx
Inferior vs inferolateralInferior = left wall in A2C, 6 o'clock in PSAX. Inferolateral = A3C / PLAX, 4 o'clock in PSAX.RCA vs LCx
Inferior vs inferoseptalInferior = A2C left wall. Inferoseptal = A4C left wall (septum). Both are usually RCA.RCA (both)

Rule of thumb

The septum in A4C is inferoseptal. The septum in PLAX / A3C is anteroseptal. The lateral wall in A4C is anterolateral. The lateral wall in PLAX / A3C is inferolateral.

Rotation sequence

A4C → (≈60° CCW) → A2C → (≈60° CCW) → A3C. Each apical plane slices the PSAX ring through two opposite walls. Tick apical planes on the basal PSAX drawing to see this.

Clock face (PSAX)

12 anterior · 2 anterolateral · 4 inferolateral · 6 inferior · 8 inferoseptal · 10 anteroseptal. The RV sits on the septal side (screen left).

Diastolic function ASE 2025 update (replaces ASE/EACVI 2016) · applies at any LVEF

Step 1: three primary variables

VariableAbnormal ifMeaning
e′ (relaxation)Septal ≤ 6 · lateral ≤ 7 · average ≤ 6.5 cm/sImpaired relaxation (age-specific values allowed)
E/e′Average ≥ 14 · septal ≥ 15 · lateral ≥ 13Filling pressure marker
TR velocity / PASPTR ≥ 2.8 m/s or PASP ≥ 35 mmHgFilling pressure marker (exclude pre-capillary PH)

Step 2: supplemental parameters (when the primary ones are discordant)

ParameterSuggests ↑ LAP if
LA reservoir strain (LARS)≤ 18 %
Pulmonary vein S/D ratio≤ 0.67 (not in young / athletes)
LA volume index (biplane)> 34 mL/m² (not in athletes, bradycardia, AF history, MR)
IVRT≤ 70 ms
OthersAr − A duration > 30 ms · Valsalva ΔE/A ≥ 50 % · mitral L-wave ≥ 50 cm/s · PR end-diastolic V ≥ 2 m/s
Figure: ASE 2025 algorithm (sinus rhythm)
Measure e′, E/e′ and TR / PASP
All normal
Normal diastolic function
normal LAP
Only e′ reduced
E/A ≤ 0.8 → Grade 1
normal LAP
E/A > 0.8 → supplemental ↓
1–2 abnormal (E/e′ or TR)
Supplemental: LARS ≤ 18 % · PV S/D ≤ 0.67 · LAVI > 34 · IVRT ≤ 70
None → normal LAP
Grade 1 if e′ ↓, else normal
≥ 1 → ↑ LAP
All 3 abnormal
↑ LAP
↑ LAP → grade by E/A: < 2 = Grade 2 (mild–moderate ↑ LAP) · ≥ 2 = Grade 3 (marked ↑ LAP)
Figure: mitral inflow (top) and septal tissue Doppler (bottom) by grade

Atrial fibrillation

Several parameters needed; no single one is reliable. ↑ LAP supported by: septal E/e′ ≥ 11 · TR > 2.8 m/s · DT ≤ 160 ms (reduced EF) · IVRT ≤ 65 ms · PV diastolic DT ≤ 220 ms · E/Vp ≥ 1.4. Average over 5–10 beats.

Special populations

MAC / MV disease: E/e′ unreliable, so use E/A and IVRT. LBBB / RV pacing: use lateral e′. Pre-capillary PH: use lateral E/e′ and LARS. Constriction: medial e′ > 7 with annulus reversus.

HFpEF diagnosis

The 2025 guideline also scores HFpEF likelihood by adding echo (↑ LAP, LA size/strain, LVH) to clinical data. Diastolic stress echo is abnormal if average E/e′ ≥ 14 (or septal ≥ 15) and peak TR > 3.2 m/s.

Atria LA: ASE/EACVI 2015 · RA: ASE 2025

ParameterNormalMildModerateSevere
LA volume index (biplane)16–3435–4142–48> 48 mL/m²
LA reservoir strain> 18 % (normal ≈ 39 %)≤ 18 % suggests ↑ LAP
LA AP diameter (PLAX)M 30–40 · F 27–38 mmDiameter underestimates size, so use volume
RA volume index (disks)< 3030–36> 36–41> 41 mL/m²
RA area (A4C, end-systole)< 1919–22> 22–24> 24 cm²
RA major dimension< 5454–58> 58–63> 63 mm
RA minor dimension< 4242–47> 47–51> 51 mm

Measure atria at end-systole (the frame before the MV/TV opens) in atrial-focused views, avoiding foreshortening. Exclude the pulmonary veins and LAA from the LA tracing. ESC 2025 counts severe LA dilatation (LAVI ≥ 60 mL/m² or diameter ≥ 55 mm) as a trigger for primary MR repair.

Right ventricle ASE 2025 right heart guideline (graded severity)

Size (RV-focused A4C, end-diastole)

ParameterNormalMildModerateSevere
RV basal diameter< 4141–44> 44–49> 49 mm
RV mid diameter< 3535–38> 38–42> 42 mm
RV longitudinal< 8282–89> 89–96> 96 mm
RVOT PLAX< 3333–35> 35–39> 39 mm
RVOT PSAX proximal< 3434–38> 38–41> 41 mm
RVOT PSAX distal< 2929–30> 30–33> 33 mm
RV wall thickness (subcostal)< 55–7> 7–9> 9 mm
RV EDA< 2525–28> 28–32> 32 cm²
RV ESA< 1414–16> 16–19> 19 cm²
3D RV EDV index< 9090–103> 103–115> 115 mL/m²
RV/LV basal ratio< 1.0> 1.0 suggests PH / RV overload

Systolic function

ParameterNormalMild ↓Moderate ↓Severe ↓
TAPSE> 1717–13< 13 to > 10≤ 10 mm
RV S′ (TDI)> 9.59.5–7.2< 7.2 to > 5.0≤ 5.0 cm/s
FAC> 3535–29< 29 to > 22≤ 22 %
RV free-wall strain (absolute)> 2020–15< 15 to ≥ 11< 11 %
RV global strain (absolute)> 1717–13< 13 to > 9≤ 9 %
3D RVEF> 4545–39< 39 to ≥ 32< 32 %
RIMP (TDI)< 0.550.55–0.610.62–0.69≥ 0.70
RIMP (PW)< 0.400.40–0.480.49–0.56≥ 0.57

RV–PA coupling

TAPSE/PASP: normal ≈ 0.5–0.7 mm/mmHg. < 0.55 is an ESC/ERS PH sign. 0.3–0.4 or lower = RV–PA uncoupling with higher mortality.

Septal flattening (PSAX)

LV eccentricity index > 1.1. D-shape in systole = pressure overload. D-shape in diastole = volume overload.

McConnell sign

RV free-wall akinesis with apical sparing suggests acute PE (not specific). "60/60" sign: PAAT < 60 ms plus TR gradient < 60 mmHg.

RV anatomy

Free wall: basal, mid, apical (A4C) · RVOT · inferior wall (subcostal). Moderator band at the apex. Septal TV leaflet sits more apical than the mitral (offset ≤ 8 mm/m²; more suggests Ebstein).

RA pressure & pulmonary hypertension

RA pressure from IVC (subcostal, 1–2 cm from RA)

IVCCollapse on sniffRAP estimate
≤ 21 mm> 50 %3 mmHg (0–5)
≤ 21 mm< 50 %8 mmHg (5–10)
> 21 mm> 50 %8 mmHg (5–10)
> 21 mm< 50 %15 mmHg (10–20)

Not valid in ventilated patients. Supportive of high RAP: restrictive TV inflow, tricuspid E/e′ > 6, diastolic-predominant hepatic vein flow, RA strain. Young athletes may have a dilated IVC with normal RAP.

Haemodynamic grading ASE 2025

ParameterNormalMildModerateSevere
RAP0–< 55–< 1010–< 15≥ 15 mmHg
TR velocity< 2.82.8–3.13.2–3.5≥ 3.6 m/s
RVSP / PASP≤ 3435–4950–69≥ 70 mmHg
RVOT acceleration time> 10580–10560–< 80≤ 60 ms
PA diameter< 2525–30> 30–35> 35 mm

Echo probability of PH ESC/ERS 2022 (PH = mPAP > 20 mmHg)

Peak TR velocityOther echo PH signsProbability
≤ 2.8 m/s or not measurableNoLow
≤ 2.8 m/s or not measurableYesIntermediate
2.9–3.4 m/sNoIntermediate
2.9–3.4 m/sYesHigh
> 3.4 m/sNot requiredHigh

A. Ventricles

RV/LV basal ratio > 1.0 · septal flattening (LVEI > 1.1) · TAPSE/PASP < 0.55 mm/mmHg

B. Pulmonary artery

RVOT AcT < 105 ms and/or mid-systolic notching · early diastolic PR velocity > 2.2 m/s · PA diameter > aortic root or > 25 mm

C. IVC & RA

IVC > 21 mm with ↓ inspiratory collapse · RA area (end-systole) > 18 cm²

"Other signs" = signs from at least 2 categories (A/B/C).

Aortic stenosis ASE/EACVI 2017 · ACC/AHA 2020 · ESC/EACTS 2025

ParameterSclerosisMildModerateSevere
Peak velocity≤ 2.5 m/s2.6–2.93.0–3.9≥ 4.0 m/s
Mean gradient—< 2020–39≥ 40 mmHg
AVA (continuity)—> 1.51.0–1.5< 1.0 cm²
AVA index—> 0.850.60–0.85< 0.6 cm²/m²
Velocity ratio (DVI)—> 0.500.25–0.50< 0.25

Very severe: Vmax ≥ 5 m/s or mean gradient ≥ 60 mmHg. Normal AVA 3–4 cm².

Figure: grading AS when the parameters disagree
AVA ≤ 1.0 cm² (AVAi ≤ 0.6)
Vmax ≥ 4 or MG ≥ 40
High-gradient severe AS
MG < 40 · SVi ≤ 35
LVEF < 50 % → classical LFLG
Dobutamine stress echo: severe if Vmax ≥ 4 / MG ≥ 40 with AVA ≤ 1.0. Flow reserve = SV ↑ ≥ 20 %
LVEF ≥ 50 % → paradoxical LFLG
CT calcium score: likely severe ≥ 2000 AU (M) / ≥ 1200 AU (F)
MG < 40 · SVi > 35
Normal-flow, low-gradient
Re-check LVOT diameter and search all windows for the peak velocity. Often moderate AS; use CT calcium if unsure

Discordant grading: check flow first

AVA < 1.0 but mean gradient < 40 mmHg. Measure SVi (≤ 35 mL/m² = low flow). Re-check the LVOT diameter first, because the error is squared.

Intervention triggers

ACC/AHA 2020: severe AS with symptoms · LVEF < 50 % · very severe (Vmax ≥ 5) asymptomatic · rapid progression (≥ 0.3 m/s/yr). ESC 2025: early intervention in asymptomatic severe AS with LVEF ≥ 50 % is now Class IIa; TAVI favoured from age ≥ 70.

Mixed aortic valve disease

ESC 2025: intervene when mean gradient ≥ 40 mmHg or Vmax ≥ 4 m/s even if the AVA is not severe, because the AR raises flow.

Aortic regurgitation ASE 2017

ParameterMildModerateSevere
Vena contracta< 0.3 cm0.3–0.6> 0.6 cm
Jet width / LVOT< 25 %25–64 %≥ 65 %
Pressure half-time> 500 ms200–500< 200 ms
Desc. aorta diastolic reversalBrief, earlyIntermediateHolodiastolic (EDV >20 cm/s)
Abdominal aorta reversal——Holodiastolic = severe
CW jet densityFaintDenseDense
Regurgitant volume< 30 mL30–59≥ 60 mL
Regurgitant fraction< 30 %30–49 %≥ 50 %
EROA< 0.10 cm²0.10–0.29≥ 0.30 cm²
LV sizeNormalNormal / dilatedDilated (chronic)

Surgery: ACC/AHA 2020 (chronic severe AR)

Symptoms · LVEF ≤ 55 % · LVESD > 50 mm or LVESDi > 25 mm/m² · progressive LVEDD > 65 mm (low risk).

Surgery: ESC 2025 (asymptomatic)

Class I: LVEF ≤ 50 % · LVESD > 50 mm · LVESDi > 25 mm/m². Consider earlier (low risk, IIb): LVESDi > 22 mm/m² · LVESVi > 45 mL/m² · LVEF ≤ 55 %. Indexing matters most when BSA < 1.68 m².

Mitral stenosis

ParameterProgressive (mild–mod)SevereVery severe
MVA (planimetry / PHT)> 1.5 cm²≤ 1.5 cm²≤ 1.0 cm²
Pressure half-time< 150 ms≥ 150 ms≥ 220 ms
Mean gradient (HR 60–80)< 5 mmHg> 5–10 (supportive)> 10
PASP< 30> 50 mmHg

MVA formulas

MVA = 220 / PHT (unreliable post-PMBC, ↓ LV compliance, severe AR) · planimetry in PSAX at leaflet tips = reference

Wilkins score (PMBC suitability)

Mobility · thickening · calcification · subvalvular — each 1–4. Total ≤ 8 favourable. Also exclude LA thrombus and > mild MR.

Rheumatic signs

Commissural fusion, "hockey-stick" AML doming, chordal thickening. Degenerative MS = MAC without commissural fusion.

Mitral regurgitation ASE 2017

ParameterMildModerateSevere
Vena contracta< 0.3 cm0.3–0.69≥ 0.7 cm
EROA (PISA)< 0.20 cm²0.20–0.39≥ 0.40 cm²
Regurgitant volume< 30 mL30–59≥ 60 mL
Regurgitant fraction< 30 %30–49 %≥ 50 %
PISA radius (Nyquist 30–40)None / smallIntermediate≥ 0.9 cm
Colour jet area (central)< 20 % LAVariable> 50 % LA / wall-hugging
Mitral inflowA-wave dominantVariableE > 1.2 m/s
Pulmonary vein flowS dominantS bluntedSystolic reversal
CW jetFaint / partialDense, parabolicDense, triangular (early peak)
StructuralNormal leaflets—Flail, ruptured papillary, large coaptation gap
Figure: PISA method
r LVLA first aliasing lineValias ≈ 30–40 cm/s vena contracta → jet

Flow = 2π r² × Valias · EROA = flow / Vmax MR · RVol = EROA × VTIMR. Shift the baseline toward the jet direction, zoom in, and measure r in mid-systole from the first aliasing line to the orifice.

Carpentier classification

I normal motion (annular dilation, perforation) · II excessive (prolapse, flail) · IIIa restricted in systole and diastole (rheumatic) · IIIb restricted in systole (ischaemic/functional)

Scallops (PSAX, lateral → medial)

A1/P1 lateral (near LAA) · A2/P2 middle · A3/P3 medial (septal). The jet points away from the prolapsing leaflet.

Secondary MR

ASE 2017 and ACC/AHA 2020 use the same thresholds (EROA ≥ 0.40). A crescent orifice makes PISA underestimate; EROA ≥ 0.20–0.30 is already prognostic. ESC 2025: TEER is Class I in selected symptomatic patients with LVEF < 50 % after optimised GDMT.

Surgery: primary severe MR

ACC/AHA 2020: symptoms · LVEF ≤ 60 % · LVESD ≥ 40 mm · new AF or PASP > 50 (reasonable). ESC 2025: also LVESDi ≥ 20 mm/m², and repair if durable when ≥ 3 of: AF · PASP > 50 · LAVI ≥ 60 mL/m² or LA ≥ 55 mm · ≥ moderate TR.

Tricuspid valve

Tricuspid regurgitation ASE 2017 + 5-grade extension

ParameterMildModerateSevereMassive / torrential
Vena contracta< 0.3 cm0.3–0.690.7–1.31.4–2.0 / ≥ 2.1 cm
EROA (PISA)< 0.20 cm²0.20–0.390.40–0.590.60–0.79 / ≥ 0.80
Regurgitant volume< 30 mL30–44≥ 45 mL
PISA radius (Nyquist ~28)≤ 0.5 cm0.6–0.9> 0.9 cm
Hepatic vein flowS dominantS bluntedSystolic reversal
CW jetFaint, parabolicDense, parabolicDense, triangular early peak
Tricuspid inflowA dominantVariableE ≥ 1.0 m/s

TV annulus > 40 mm (or > 21 mm/m²) in A4C diastole → consider TV annuloplasty at time of left-sided surgery. Leaflets: anterior, septal, posterior.

Tricuspid stenosis — severe

Mean gradient ≥ 5 mmHg · PHT ≥ 190 ms · TVA ≤ 1.0 cm² (continuity) · inflow VTI > 60 cm.

Pulmonic valve

Pulmonic stenosis

ParameterMildModerateSevere
Peak velocity< 3 m/s3–4 m/s> 4 m/s
Peak gradient< 36 mmHg36–64> 64 mmHg

Pulmonic regurgitation

ParameterMildModerateSevere
Jet width / annulusSmall, narrow originIntermediate> 70 %
CW jet / PHTFaint, slow decel.Dense, variableDense, steep; PHT < 100 ms
PR index——< 0.77
Regurgitant fraction (CMR)< 20 %20–40 %> 40 %
Branch PA flow——Diastolic flow reversal

PR end-diastolic velocity → PADP = 4·V² + RAP. Early PR velocity → mPAP ≈ 4·V² + RAP.

Prosthetic valves ASE 2024 prosthetic valve guideline

Aortic prosthesis

ParameterNormalPossible stenosisSignificant stenosis
Peak velocity< 3 m/s3–4> 4 m/s
Mean gradient< 20 mmHg20–34≥ 35 mmHg
DVI≥ 0.300.25–0.29< 0.25
EOAWithin reference for model/size< reference − 1 SD< reference − 2 SD
Acceleration time< 80 ms80–100> 100 ms
AT / ET< 0.320.32–0.37> 0.37
Jet contourTriangular, early peakIntermediateRounded, symmetric

Patient–prosthesis mismatch (aortic, indexed EOA)

BMINo / insignificantModerateSevere
< 30 kg/m²> 0.850.66–0.85≤ 0.65 cm²/m²
≥ 30 kg/m²> 0.700.56–0.70≤ 0.55 cm²/m²

Mitral prosthesis

ParameterNormalPossible stenosisSignificant stenosis
Peak E velocity< 1.9 m/s1.9–2.5≥ 2.5 m/s
Mean gradient≤ 5 mmHg6–10> 10 mmHg
VTIPrMV / VTILVOT< 2.22.2–2.5> 2.5
EOA≥ 2.0 cm²1–2< 1 cm²
PHT< 130 ms130–200> 200 ms

Reading a high gradient

High gradient with a normal DVI / VTI ratio → high flow or PPM. High gradient with an abnormal ratio → obstruction (thrombus or pannus), or significant MR for a mitral prosthesis. High velocity with short AT (< 80 ms) → pressure recovery or small valve, not obstruction.

Paravalvular regurgitation

Circumferential extent of the sewing ring: < 10 % mild · 10–29 % moderate · ≥ 30 % severe. Combine with jet-based and volumetric parameters.

Baseline study

Get a TTE 6 weeks to 3 months after implantation as the patient's own reference ("fingerprint"). Compare every later study with it.

Aorta measure leading-edge to leading-edge, end-diastole (annulus: mid-systole, inner-inner)

LevelMen (mean ± SD)Women (mean ± SD)≈ Upper normal
Annulus26 ± 3 mm23 ± 2 mm32 / 27 mm
Sinus of Valsalva34 ± 3 mm30 ± 3 mm40 / 36 mm
Sinotubular junction29 ± 3 mm26 ± 3 mm35 / 32 mm
Proximal ascending30 ± 4 mm27 ± 4 mm38 / 35 mm

Size depends on BSA, age and sex — prefer indexed values / z-scores. Surgery (ACC/AHA 2022): root/ascending ≥ 55 mm; ≥ 50 mm in selected (e.g. bicuspid with risk factors, low surgical risk); ≥ 45 mm if undergoing other cardiac surgery; Marfan ≥ 50 mm (≥ 45 with risk factors).

Pericardium

Effusion (end-diastole, echo-free space)Size
Only in systoleTrivial
< 10 mmSmall
10–20 mmModerate
> 20 mmLarge

Tamponade

RA systolic collapse > 1/3 of cycle · RV diastolic collapse · plethoric IVC without collapse · mitral inflow respiratory variation > 25–30 % · tricuspid > 40–60 % · swinging heart. Clinical diagnosis.

Constriction

Respiratory septal shift / bounce · medial e' ≥ 9 cm/s (annulus reversus: medial > lateral) · hepatic vein expiratory diastolic reversal (ratio ≥ 0.79) · mitral E variation > 25 % · thick pericardium. Mayo: septal shift + medial e' ≥9 + HV reversal ≥0.79.

Effusion vs pleural (PLAX)

Pericardial fluid tracks anterior to descending aorta; pleural fluid posterior to it. Epicardial fat: anterior, speckled, moves with heart.

Hypertrophic cardiomyopathy & others AHA/ACC 2024 HCM guideline

HCM

Maximum wall thickness ≥ 15 mm (≥ 13 mm with family history or positive genotype) not explained by loading. Measure every segment in PSAX. ≥ 30 mm is an SCD risk factor, as are an apical aneurysm and LVEF < 50 %.

LVOT obstruction

Peak gradient ≥ 30 mmHg (rest or provoked) = obstruction · ≥ 50 mmHg = haemodynamically significant (septal reduction / myosin-inhibitor threshold). Provoke with Valsalva, standing or exercise. Late-peaking "dagger" CW. SAM ± posteriorly directed MR.

Myosin inhibitors (mavacamten)

Echo monitoring of LVEF and LVOT gradient. Interrupt treatment if LVEF < 50 %.

Amyloid red flags

↑ wall thickness with low QRS voltage · granular sparkle · biatrial enlargement · small effusion · restrictive filling · apical-sparing strain (relative apical LS ratio > 1) · low LVOT SV despite "normal" EF.

LV thrombus

Apical akinesis or aneurysm · echodense mass distinct from the endocardium, seen in 2 views · use contrast if unclear.

Formulas

Haemodynamics

ΔP = 4 · V² (simplified Bernoulli) LVOT area = 0.785 · d² SV = LVOT area × VTILVOT CO = SV × HR Qp/Qs = (d²RVOT·VTIRVOT) / (d²LVOT·VTILVOT) (>1.5 significant)

Pressures

PASP = 4·TRV² + RAP mPAP ≈ 4·(early PR V)² + RAP PADP = 4·(end PR V)² + RAP mPAP ≈ 79 − 0.45·AcT PVR ≈ 10·TRV/VTIRVOT + 0.16 WU (ratio >0.2 → PVR >2) LAP ≈ 1.24·(E/e') + 1.9 LVEDP ≈ DBP − 4·(end-diastolic AR V)²

Valves

AVA = (LVOT area × VTILVOT) / VTIAV DVI = VTILVOT / VTIAV MVA = 220 / PHT PISA flow = 2π·r²·Valiasing EROA = PISA flow / Vmax; RVol = EROA × VTIreg Quick MR EROA ≈ r²/2 (Nyquist 40, MR V ≈ 5 m/s)

LV

LV mass = 0.8·1.04·[(IVS+LVID+PW)³ − LVID³] + 0.6 g (cm) RWT = 2·PWd / LVIDd FS = (LVIDd − LVIDs) / LVIDd EF = (EDV − ESV) / EDV dP/dt = 32 mmHg / Δt(1→3 m/s on MR CW) MPI = (IVCT + IVRT) / ET BSA (Mosteller) = √(ht cm × wt kg / 3600)
Summary
➜